What is Pulmonary Fibrosis?
Pulmonary fibrosis is a chronic and progressive lung disease where the air sacs in the lungs, called the alveoli, become scarred and stiff, making it difficult to breathe and get enough oxygen into the bloodstream.
According to the British Lung Foundation – Pulmonary fibrosis is an interstitial lung disease (ILD). There are more than 200 different ILDs.
- pulmonary: it affects your lungs
- fibrosis: a build-up of scar tissue, which makes your lungs stiff
- interstitial: affects your interstitium, the network of tissue that supports the air sacs in your lungs
What Causes Pulmonary Fibrosis?
There are a number of known causes of pulmonary fibrosis as well as unknown causes, called idiopathic. Exposure to toxins like asbestos, or coal dust or silica (including workers in the coal mining and sandblasting industry) can lead to pulmonary fibrosis. There are medications known to have a side effect of pulmonary fibrosis (amiodarone, bleomycin, nitrofurantoin, to name a few). A more common cause of pulmonary fibrosis is that seen in conjunction with a group of diseases known as collagen vascular diseases. This would include systemic lupus, scleroderma, rheumatoid arthritis and Sjogren’s syndrome. There can be familial or hereditary cases of pulmonary fibrosis as well. And as mentioned earlier, there are many cases of pulmonary fibrosis which cannot currently identify a cause and is therefore called “idiopathic” pulmonary fibrosis.
Reversal by Providing the Missing microRNAs.
Recent observations indicate that fibrosis, aging and abnormal wound healing are linked to the down expression of specific genes. More specifically, researchers have shown that the expression of MicroRNAs of the miR-29 family are significantly reduced in fibrotic lungs. They show that miR-29 levels are inversely correlated with the severity of the fibrosis. ( Am. J. Respir. Cell. Mol. Biol. 2011 Aug) Embryonic stem cells express miR29 as can be seen in this extensive publication of the microRNAs released by them, For this reason we first observed reversal of keloids on wounds as a positive side-effect of our anti-aging stem cell treatment. Then we observed that fibrotic livers and lungs have improved function following our stem cell therapy.
Our fibrosis reversal protocol includes a recommended minimum dose of 6 stem cell injections to initiate the reversal process, following by regular “maintenance” injections. For more information,
It is now understood that most forms of COPD/ emphysema result from accelerated ageing, a pathological mechanism also involved in various chronic disease. Genes associated with inflammation are upregulated in COPD sufferers while genes associated with tissue repair are down-regulated. The leucocyte telomere lengths, one of the main ageing markers, are also much lower than average for COPD patients and their lung fibroblasts have impaired ability to restructure collagen.
Benefits of Embryonic Stem Cells for Pulmonary Fibrosis Treatment
atient Data: Our most recent pulmonary fibrosis patient had been diagnosed with a progressive form of the disease over one year ago. Here is his progress report after having received 10 Stem Cell Health Embryonic Stem Cell injections.
Feeling fantastic. Arthritic pain in my neck and back has cleared up completely. I am looking younger.
His oxygenation capacity started to improve following his third injection. The Dupuytren’s contracture is linked to the same fibrosis condition and can also be reversed with embryonic stem cell therapy.
Our fibrosis reversal protocol includes a recommended minimum dose of 6 stem cell injections to initiate the reversal process, following by regular “maintenance” injections.
To learn more about our protocol or to be directed to an affiliated practitioner, Click the “ask a question” button below.
Contact Stem Cell Health to learn more about innovative stem cell therapy.